The criticism that bacteria and/or viruses are incapable of surviving under the harsh conditions of space is certainly not borne out by all the data that has accumulated over the past three decades. Bacteria and viruses embedded in minute grains of rock, dust, carbonaceous material, or ice, are protected effectively from radiation damage and can remain fully viable for millions of years under space conditions.
If you’re interested in Covid from Space, this is your summer reading. While it is preceded by a variety of other recent journal articles (many hundreds if you include the wider literature of Panspermia) these four publications are the starting point for truly understanding Covid. They tell us more about the disease in one fell swoop than all the other global science pubs since January 2020 combined.
But they don’t tell us everything. To learn the rest of the story — or anything approaching it — these papers will need to be “accepted” as legitimate despite their revolutionary thinking. Nothing much of relative importance can be further discovered about Covid-19 unless and until you start with the work right here of Steele, Wickramsinghe, et al.
Despite a half century of Hoyle and Wickramasinghes suppression, and the large swath of experts and their disciplines that would be permanently humiliated, the Tusk believes the recent papers will be accepted — and sooner rather than later.
The arc of science bends towards truth. And in this case perhaps quickly, if one puts aside the previous five decades of delay.
Whilst there is certainly no groundswell of disease from space adherents, Panspermia in general is being verified on a regular and increasingly frequent basis using modern instruments and long delayed experiments. It will not be too long now before the frustration with Covid ignorance, and global surprise by overwhelming evidence for the omnipotence of dessicated and hibernated microbial life in space, will converge to overturn Aristotle and Darwin’s warm little pond.
And at long last we will reposition the earth as a part — not the start — of life.
A quick summary of the science from Dr. Ted Steele:
Our explanation is that the virus arrived as fragmented meteorite dust in the stratosphere over China (Oct 11 2019), striking Wuhan within a few weeks, then much of this dust load was then caught in in the upper troposphere/lower stratosphere Jet-Streams and taken East to West coming down in quick succession in Tehran/Qom, Lombardy/Italy, Spain and then New York City ( by early- to mid March 2020). The latter NYC strike we predicted in advance as this East-> West progress was along the 40o N Latitude band. The viral laden dust clouds (bearing the source COVID-19 virions) have since fragmented and become patchy striking many European and North American regions, and the dust clouds have also entered the Southern hemisphere (Brazil, much of South America, South Africa, Australia, and New Zealand, and currently engaging French Polynesia. But other Asian countries are also engaged (Pakistan, India, recently Sri Lanka) and also many other regions, but it seems to a variable lesser extent from public case records (Japan, South Korea, Taiwan, Philippines)
Thus we are scientific experts that bring together a coherent string of causal evidence covering the totality of factual data at all key levels: genetic, immunologic, epidemiologic, geophysical, astrophysical and astrobiological explanations as this pandemic has unfolded in real-time. Our papers can be read as a chronological scientific testimony of the evidence supporting this explanation (Two of our earlier papers just prior to the pandemic ironically also “predicted” such a Panspermic event and sets the stage for our ability to track the virus- see last two reference URLs below).
So the virus is coming in dust clouds from the air, carried in variable size meteorite and then pollution dust particles, each particle (say the dangerous small 2.5 micron sizes) can be expected to carry an infective minimum dose of a cluster of (say) 10-50 COVID-19 virions. This is COVID-19’s primary mode of spread, drifting and coming down as dust particles from the stratosphere/troposphere.
The environment where a viral-laden dust cloud is brought down (mainly by rain) is then heavily contaminated (we believe left unattended the virus will decay in most environments across the planet over 2-3 months). People primarily get the virus by “rubbing up against their contaminated environment” and, in some cases, breathing the air at in-fall (so masks may help during that interval).
Person-to-Person (P-to-P) spread clearly occurs but this spread is secondary to the main in-fall features and signatures (e.g. why passengers, crew, and military personal on ships at sea are vulnerable; the high incidence of “mystery infections “ or community spreads without a known source in Victorian 2nd Wave, n ≥ 4100 cases).
But P-to-P is a very clear feature of closed nursing homes and aged care facilities with many elderly co-morbids (who really needed to be protected and were not) – in Victoria, younger and healthy carers who were mildly affected and asymptomatic working in multiple facilities at low wages were the primary spreading vector (it seems from all news reports here in Victoria).
The virus has a clear haplotype switching strategy allowing it to sort the ‘best’ haplotype for growth in that genetic/biochemical background (read broadly as “ethnic” group) – in an average productive infection maybe 2-6 mutations may accrue across the approx. 30,000 nucleotide length genome. The most rampant P-to-P however is in the aged co-morbids in closed facilities (also cruise ships).
Thus we can provide all the key scientific evidence on COVID-19 origins. The medical and scientific advisors to the government have the completely wrong infection model (P-to-P exclusively) in their collective heads.
There is another assumption about the origins of the COVID-19 virus that can be disposed of.
In a key paper we address the popular myth that COVID-19 and other diseases like influenza are zoonotic, i.e. they are emerging constantly from infected animals (zoonotic diseases). These claims are not actually supported by any direct evidence, and the evidence that is available bearing on the issue is actually very weak and certainly does not support this mainstream conception.
In the case of COVID-19 our genetic analysis of all relevant current data shows that such an animal-to-human jump (on available existing data) has at the bear minimum one chance in 10180of being successful – for these odds the starting point is from a fictitious closest known animal-sourced relative that may be 99% similar in sequence to COVID-19 (the Steele, Gorczynski et al 2020 paper is on the list and is athttps://www.hilarispublisher.com/open-access/implications-of-haplotype-switching-for-the-origin-and-global-spread-of-covid19.pdf ). The closest known bat relative is 96.2% similar in sequence to COVID-19 and for that to jump and be a successful complete match to COVID-19 the odds blow out to one successful trial in10684. These are impossible odds, far beyond the molecular resources of our known universe viz. ~ 1084 protons or atomic nuclei.
In the case of a detailed attempted experimental reconstruction of the 2009 H1NI swine flu pandemic, on the key responsible H1N1 strain, the actual experimental data† showed that such a complex re-assortment of flu genetic segments from precursor/donor strain does not in fact happen easily in multiply infected pigs.
Papers on Origin and Global Spread of COVID-19 : Wickramasinghe, Steele, Gorczynski, Lindley, Tokoro, Temple, Rebhan, Howard, Carnegie, Kondakov, Klyce, Dayal Wickramsinghe, Coulson, Wallis (Max), Wallis (Daryl), Ananda Nimalasuriya,Milton Wainwright
Wickramasinghe, Steele , Gorczynski et al 2020 Virology Current Research
Comments on the Origin and Spread of the 2019 Coronavirus
Wickramasinghe, Steele , Gorczynski et al (2020) Virology Current Research
Growing Evidence against Global Infection-Driven by Person-to-Person Transfer of COVID-19. Virology Current Research Volume 4:1,2020DOI: 10.37421/Virol Curr Res.2020.4.110
Wickramasinghe, Steele, Gorczynski et al (2020) Virology Current Research Predicting the Future Trajectory of COVID-19. Virology Current Research Volume 4:1,2020 DOI DOI: 10.37421/Virol Curr Res.2020.4.111
Howard, GA, Wickramasinghe, NC, Rebhan, H et al (2020) Mid-Ocean Outbreaks of COVID-19 with Tell-Tale Signs of Aerial Incidence Virology Current Research Volume 4:1,2020 DOI: 10.37421/Virol Curr Res.2020.4.114
Steele, E.J., Gorczynski ,R.M,. Rebhan, H., Carnegie, P., Temple, R., Tokoro, G., et al (2020) Implications of haplotype switching for the origin and global spread of COVID-19 Virol Curr Res Volume 4:2, 2020 DOI: 10.37421/Virol Curr Res.2020.4.115
Steele, E.J ., & Lindley, R.A .(2020) Analysis of APOBEC and ADAR deaminase-driven Riboswitch Haplotypes in COVID-19 RNA strain variants and the implications for vaccine design. Research Reports doi:10.9777/rr.2020.10001
Some of our Cosmic Genetic Evolution Papers just prior to COVID-19
Steele EJ, Al-Mufti S, Augustyn KK, Chandrajith R, Coghlan JP, Coulson SG, Ghosh S, Gillman M. et al 2018 “Cause of Cambrian Explosion: Terrestrial or Cosmic?” Prog. Biophys. Mol. Biol. 136: 3-23, https://doi.org/10.1016/j.pbiomolbio.2018.03.004
Steele EJ, Gorczynski RM, Lindley RA, Liu Y, Temple R, Tokoro G, Wickramasinghe DT, Wickramasinghe NC. 2019 “Lamarck and Panspermia – On the Efficient Spread of Living Systems Throughout the Cosmos”. Prog. Biophys. Mol. Biol. 2019 149 : 10 -32.